Zitat:
Mente Autism hilft den Kindern, ihr Gehirn zu entspannen und sich so besser zu konzentrieren und zu fokussieren. Weil es ihnen leichter fällt, mit anderen Menschen umzugehen, verbessern sich bei vielen auch die schulischen Leistungen.
Zitat:
Mit einer speziell für autistische Kinder entwickelten Behandlung, die auf bewährten Neurofeedback-Methoden beruht, werden die Kinder innerhalb kurzer Zeit ruhiger, aufmerksamer und konzentrierter. Sie können besser kommunizieren und mit anderen Menschen interagieren.
Zitat:
Alpha-, Beta-, Delta- und Theta-Wellen werden normalisiert
Bei Autisten sind häufig die Alpha- und Beta-Wellen des Gehirns niedriger und die Delta- und Theta-Wellen höher als durchschnittlich. Solche Aktivitätsmuster werden bei gesunden Menschen in Schlafphasen und bei geschlossenen Augen sowie bei Tagträumerei gemessen. Im wachen, aufmerksamen und konzentrierten Zustand zeigen die Gehirnströme ein völlig anderes Muster. Mit der auditiven Neurofeedback-Therapie mit Mente Autism werden Alpha-, Beta-, Delta-und Thetawellen der betroffenen Kinder aufgezeichnet und analysiert. Die spezielle Software wandelt diese Aufzeichnung in ganz besondere, individuelle Töne um, die wiederum die Gehirnwellen beeinflussen:
Alpha- und Beta-Wellen sollen dadurch höher, Delta- und Theta-Wellen niedriger werden. Die Kinder werden aufmerksamer, wacher und kommunikationsfähiger.
Zitat:
Cantor and colleagues [72] reported that subjects with ASD had enhanced power in the delta band, in the posterior-temporal, midline, and occipital regions of the left hemisphere. Similarly, Stroganova et al. [74] found enhanced delta power in the left hemisphere of individuals with ASD in the frontal, temporal, and parietal regions. In the theta band, left-hemisphere dominance in ASD was seen in frontal [74,78], parietal [74], temporal [72,74], and occipital [72,74] regions. In the alpha band, left-hemisphere dominance in ASD was reported in multiple studies in mid-frontal [106,107], temporal, parietal [72,74], midline [72,106], and occipital [72] regions. Finally, Cantor et al. [72] replicated the left-hemisphere dominance pattern in the beta band in posterior-temporal, midline, and occipital regions.
Left-hemisphere asymmetry in ASD is of clinical interest, given the common language abnormalities seen in ASD [108-111]. Increased resting power in the left hemisphere may contribute to left-hemisphere performance deficits by decreasing the signal-to-noise ratio during active tasks, similar to reports of increased background noise and behavioral performance impairment in the literature on schizophrenia [112,113]. Left-hemisphere dysfunction may also be dependent on the task that subjects are performing. When performing tasks of executive functioning (for example, Go/No-go and Stroop tests), high-functioning adults with ASD had significantly increased activation restricted to the left hemisphere [114]. Left-hemisphere dysfunction has also been identified in smooth pursuit eye movements in individuals with ASD [115], as have left-lateralized alterations during an oculomotor serial reaction time task [116].
Nevertheless, as in many neuropsychiatric disorders, evidence of lateralized abnormalities has been inconsistent [74]. Dawson et al. [80] reported reduced delta power in the left mid-temporal cortex, and Lazarev et al. [105] reported no left/right-hemisphere differences frequency bands in the occipital cortex. However, in the same and subsequent reports, the same authors noted hyperconnectivity within the left hemisphere [117] and reduced power in the right hemisphere [105] in children with ASD during presentation of photic driving stimulation. Dawson and colleagues [80] utilized a relatively short (1 second) window to measure delta power, which may affect reliability of measured low-frequency activity. Lazarev and colleagues [105,117] measured activity only in 14 relatively heterogeneous subjects with ASD, so those studies may have lacked statistical power to detect effects. Photic driving is a robust response, and would potentially be more sensitive to small pathological alterations in studies with small patient cohorts.